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SDF1α-induced chemotaxis of JAK2-V617F-positive cells is dependent on Bruton tyrosine kinase and its downstream targets PI3K/AKT, PLCγ1 and RhoA

Department of Hematology and Oncology, Medical Center, Otto-von-Guericke University, Magdeburg;Health Campus Immunology, Infectiology and Inflammation, Otto-von-Guericke-University, Magdeburg;Medical Clinic and Polyclinic III, Klinikum rechts der Isar, Technical University Munich
Department of Hematology and Oncology, Medical Center, Otto-von-Guericke University, Magdeburg;Health Campus Immunology, Infectiology and Inflammation, Otto-von-Guericke-University, Magdeburg
Department of Hematology and Oncology, Medical Center, Otto-von-Guericke University, Magdeburg;Health Campus Immunology, Infectiology and Inflammation, Otto-von-Guericke-University, Magdeburg
Department of Hematology and Oncology, Medical Center, Otto-von-Guericke University, Magdeburg;Health Campus Immunology, Infectiology and Inflammation, Otto-von-Guericke-University, Magdeburg
Department of Cardiology and Angiology, Medical Center, Otto-von-Guericke University, Magdeburg
Medical Clinic and Polyclinic III, Klinikum rechts der Isar, Technical University Munich;Department of Hematology, Oncology and Tumor Immunology, Campus Benjamin Franklin, Charité -Universitätsmedizin, Berlin;German Cancer Research Center (DKFZ) and German Cancer Consortium (DKTK), Heidelberg, Germany
Department of Hematology and Oncology, Medical Center, Otto-von-Guericke University, Magdeburg;Health Campus Immunology, Infectiology and Inflammation, Otto-von-Guericke-University, Magdeburg
Department of Hematology and Oncology, Medical Center, Otto-von-Guericke University, Magdeburg;Health Campus Immunology, Infectiology and Inflammation, Otto-von-Guericke-University, Magdeburg
Vol. 104 No. 7 (2019): July, 2019 https://doi.org/10.3324/haematol.2018.201921